Creatine supplements helped cancer spread in mice, but the reported human findings do not establish that outcome in people. The study, published in Nature Communications, tested several mouse models and healthy volunteers. It focused on Platelets, the small blood parts that help form clots. These became more active after creatine supplementation, including in the healthy volunteers. In mice, added creatine changed the cells that make these blood parts. It increased creatine kinase B, a protein that helps cells use energy. That triggered signals that made the clotting cells more active and helped cancer spread. Blocking parts of this process stopped the extra activity and cancer spread in mice. The human result needs careful reading. The volunteers showed increased activity in their clotting cells. The supplied summary does not report cancer spreading in those volunteers. It also gives no doses, study length, group sizes, or risk estimates. Those gaps limit what we can say about everyday supplement use. For members tracking Creatine Kinase (CK), an important distinction matters here. The study examined a specific protein inside cells, not routine blood test results. It does not show that a higher result on your bloodwork signals cancer spread. If you log Creatine Monohydrate, this offers a reason for thoughtful community discussion—not a way to judge cancer risk from your numbers. Comparing supplement logs, workout dates, and blood test timing can give shared results useful context. Hard workouts can raise this muscle marker on their own. What patterns have you noticed in your Creatine Kinase (CK) results alongside supplement use and workout timing?